Nutrient status markers is closer to a research marker, so it should be read separately from a directly felt benefit.
Representative tier calculated from paper evidence that passed the collection audit.
The representative ingredient tier is calculated from these target-level evidence groups.
10 new papers were added in this period. No new risk signal was identified.
Standalone side-effect signals and combination cautions are listed separately.
No standalone side-effect or combination signal is currently clear enough to show from the collected papers. This does not mean there is no concern.
Paper IDs and full lists are private. Only study types and summaries are shown.
There was no consistent association of urinary sodium, potassium, or sodium/potassium ratio with CVD and all-cause mortality over the range of intakes observed in this population, and the effect of sodium and potassium intake on CVD morbidity and mortality in
A simple scaling theory is derived that explains this anomalous dependence of the decay length on the ion concentration for concentrated electrolytes and can be an order of magnitude larger than the ion diameter in ionic liquids.
In patients with T2DM, canagliflozin was generally associated with small mean percent changes in serum electrolytes, and in patients taking anti-hypertensive agents that affect potassium excretion in both the canag liflozin and placebo groups.
The positive impact on dialysis efficiency, acid-base status and haemodynamics, as well as the subjective tolerance, together indicate that citrate dialysate can significantly contribute to improving haemodialysis in selected patients.
Urinary sodium excretion correlates with elevated blood pressure in subjects at low cardiovascular risk and no pattern of increased cardiovascular disease, heart failure, or mortality risk was demonstrated with either high or low sodium intake.
The findings suggest that the skin may buffer dietary Na+, reducing the hemodynamic consequences of increased salt, and this may be influenced by sex.